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It is evident that the inflammatory response and oxidative stress induced by dysbiosis not only increase the levels of pro-inflammatory cytokines but also enhance the expression of biochemical markers related to oxidative stress, thereby further accelerating the degradation of fetal membranes

doi: 10.1007/s00280-021-04347-6 202

This pattern repeats across multiple outcome measures and has direct implications for how a responsible dosing protocol should be structured

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