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There is also no significant difference in the percentage of subjects responding to GLP-1 M with or without NSAID usage (Fig

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659 The level of GPX4 in the brain gradually decreases after TBI, and overexpression of GPX4 can partially reverse TBI-induced hippocampal ferroptosis and synaptic damage, effectively alleviating neuronal dysfunction after cerebral hemorrhage, and improving TBI-induced cognitive deficits

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