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The authors' conclusion

Following the finding that the cancer cell lines did not elevate telomerase enzyme activity upon epitalon treatment but did elongate telomeres, we investigated the other commonly used mechanism to extend telomeres in mammals, ALT (Alternative Lengthening of Telomeres)

Dicks et al., 2020), and its chronology in the neurodegenerative cascade (preceded by inflammation, oxidative stress, vascular dysregulation, and A deposition) (Iturria-Medina et al., 2016), measures of brain structure can provide tangible insight into whether exercise as a therapeutic intervention is efficacious for slowing the rate of atrophy with age and neurodegenerative disease

Conversely, colonization of mice with certain commensals from healthy human guts can protect against EAE [185]