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10.1016/j.ejphar.2023.175746 60 HuangX

Further supporting this, Lock and colleagues demonstrated that glutathione protects against 2-chloropropionic acid (CPA)-induced cerebellar toxicity by preserving thiol redox equilibrium and potentially interacting with NMDA receptor-mediated pathways

It is also recognized for its ability to reduce neuroinflammation, offering benefits for individuals with cognitive decline, neurodegenerative conditions, or brain injuries

However, the test does identify individual metabolic traits that may influence overall treatment optimization and side effect tolerance

ChiappelliJRowlandLMWijtenburgSAMuellerkleinFTagametsMMcmahonRPet al