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Targeting SRC coactivators blocks the tumor-initiating capacity of cancer stem-like cells

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doi: 10.3389/fendo.2022.920541 Received 14 April 2022 Accepted 19 August 2022 Published 08 September 2022 Volume 13 - 2022 Edited by Rishi Raj, Pikeville Medical Center, United States Reviewed by Sudhanshu Kumar Bharti, Patna University, India

Furthermore, the mutator phenotype associated with defective MutL activity was suppressed when a non-functional recB allele, derived from ET-37 meningococcal strains, replaced the functional recB allele, suggesting that in MutL-deficient strains, hypermutation mostly arises during post-replicative DNA synthesis associated with the activity of the RecBCD recombination pathway (Colicchio et al., 2006a)

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