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IL-1 also promotes the release of danger signals such as HMGB1 from damaged cells, further amplifying the inflammatory response via the TLR4 pathway (discussed later in the mechanism of HMGB1) (Phan et al., 2022

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The interest in exploring this potential connection stems from emerging research into the anti-inflammatory properties of GLP-1 signaling and observations in patients with coexisting metabolic and inflammatory conditions