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Increased cAMP upregulates protein kinase A (PKA) and exchange protein activated by cAMP, which leads to rapid increases in intracellular calcium and insulin exocytosis in a glucose-dependent manner [33]

[18] [19] Treatment typically involves a combination of non-pharmacological and pharmacological interventions, tailored to the individual's age, symptom severity, and personal circumstances

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Allosteric (off active site) activators were reported in the literature as early as the mid-2000s, but they had poor potency, poor pharmacokinetics, species-selective activity (human but not rodent), and none made it close to the clinic